https://erowid.org/archive/rhodium/chemistry/pcp/effects.htmlAt low to moderate doses, the physiological effects of PCP may include a slight increase in breathing rate and a more pronounced rise in blood pressure and pulse rate. Respiration becomes shallow, and flushing and profuse sweating may occur. Generalized numbness of the extremities and muscular incoordination are common. The pupils are neither constricted nor dilated. Nystagmus (involuntary movement of the eyes) is a characteristic effect, and may be vertical, rotary, or horizontal. Motor symptoms are common and may include ataxia (impaired coordination of movement), athetosis (slow writhing movements), myoclonic jerks, and bizarre posture.At higher doses, there is a drop in blood pressure, pulse rate, and respiration. This may be accompanied by nausea, vomiting, blurred vision, extreme nystagmus, drooling, loss of balance, and dizziness. High doses of PCP can cause seizures, coma, and death (though death more often results from accidental injury or suicide during PCP intoxication).Treatment of PCP overdose is largely symptomatic and centers around increasing elimination. PCP is eliminated in the GI tract and then quickly reabsorbed, resulting in very long lasting effects. Oral administration of activated charcoal can therefore be helpful to bind it after elimination and remove it from circulation. Increasing the urine acidity by administration of ammonium chloride (or by drinking cranberry juice) also speeds elimination. If vital signs are stable and the primary symptom is confusion or agitation, it is best to reduce all sensory stimulation (such as touching and sound) as such stimulation may lead to an increase in disorientation. Agitation may be treated with valium. Phenothiazines such as haloperidol have been shown to decrease recovery time, but they may carry the increased risk of siezures (ref. 18).